PMID- 22780564
OWN - NLM
STAT- MEDLINE
DCOM- 20121121
LR  - 20240318
IS  - 1467-789X (Electronic)
IS  - 1467-7881 (Print)
IS  - 1467-7881 (Linking)
VI  - 13
IP  - 10
DP  - 2012 Oct
TI  - Impact of postprandial glycaemia on health and prevention of disease.
PG  - 923-84
LID - 10.1111/j.1467-789X.2012.01011.x [doi]
AB  - Postprandial glucose, together with related hyperinsulinemia and lipidaemia, has 
      been implicated in the development of chronic metabolic diseases like obesity, 
      type 2 diabetes mellitus (T2DM) and cardiovascular disease (CVD). In this review, 
      available evidence is discussed on postprandial glucose in relation to body 
      weight control, the development of oxidative stress, T2DM, and CVD and in 
      maintaining optimal exercise and cognitive performance. There is mechanistic 
      evidence linking postprandial glycaemia or glycaemic variability to the 
      development of these conditions or in the impairment in cognitive and exercise 
      performance. Nevertheless, postprandial glycaemia is interrelated with many other 
      (risk) factors as well as to fasting glucose. In many studies, meal-related 
      glycaemic response is not sufficiently characterized, or the methodology with 
      respect to the description of food or meal composition, or the duration of the 
      measurement of postprandial glycaemia is limited. It is evident that more 
      randomized controlled dietary intervention trials using effective low vs. high 
      glucose response diets are necessary in order to draw more definite conclusions 
      on the role of postprandial glycaemia in relation to health and disease. Also of 
      importance is the evaluation of the potential role of the time course of 
      postprandial glycaemia.
CI  - (c) 2012 ILSI Europe. obesity reviews (c) 2012 International Association for the 
      Study of Obesity.
FAU - Blaak, E E
AU  - Blaak EE
AD  - Department of Human Biology, School of Nutrition & Toxicology Research and 
      Metabolism (NUTRIM), Maastricht University, Maastricht, the Netherlands.
FAU - Antoine, J-M
AU  - Antoine JM
FAU - Benton, D
AU  - Benton D
FAU - Bjorck, I
AU  - Bjorck I
FAU - Bozzetto, L
AU  - Bozzetto L
FAU - Brouns, F
AU  - Brouns F
FAU - Diamant, M
AU  - Diamant M
FAU - Dye, L
AU  - Dye L
FAU - Hulshof, T
AU  - Hulshof T
FAU - Holst, J J
AU  - Holst JJ
FAU - Lamport, D J
AU  - Lamport DJ
FAU - Laville, M
AU  - Laville M
FAU - Lawton, C L
AU  - Lawton CL
FAU - Meheust, A
AU  - Meheust A
FAU - Nilson, A
AU  - Nilson A
FAU - Normand, S
AU  - Normand S
FAU - Rivellese, A A
AU  - Rivellese AA
FAU - Theis, S
AU  - Theis S
FAU - Torekov, S S
AU  - Torekov SS
FAU - Vinoy, S
AU  - Vinoy S
LA  - eng
PT  - Journal Article
PT  - Review
DEP - 20120711
PL  - England
TA  - Obes Rev
JT  - Obesity reviews : an official journal of the International Association for the 
      Study of Obesity
JID - 100897395
RN  - 0 (Blood Glucose)
RN  - 0 (Incretins)
SB  - IM
MH  - Blood Glucose/metabolism
MH  - Body Weight/*physiology
MH  - Chronic Disease/*epidemiology/prevention & control
MH  - Humans
MH  - Hyperglycemia/metabolism/*physiopathology
MH  - Incretins/metabolism
MH  - Obesity/*metabolism
MH  - Postprandial Period
MH  - Risk Factors
PMC - PMC3494382
EDAT- 2012/07/12 06:00
MHDA- 2012/12/10 06:00
CRDT- 2012/07/12 06:00
PHST- 2012/07/12 06:00 [entrez]
PHST- 2012/07/12 06:00 [pubmed]
PHST- 2012/12/10 06:00 [medline]
AID - 10.1111/j.1467-789X.2012.01011.x [doi]
PST - ppublish
SO  - Obes Rev. 2012 Oct;13(10):923-84. doi: 10.1111/j.1467-789X.2012.01011.x. Epub 
      2012 Jul 11.