PMID- 28001190
OWN - NLM
STAT- MEDLINE
DCOM- 20180129
LR  - 20180129
IS  - 2175-8239 (Electronic)
IS  - 0101-2800 (Linking)
VI  - 38
IP  - 4
DP  - 2016 Dec
TI  - An update on pathology of IgA nephropathy.
PG  - 435-440
LID - S0101-28002016000400435 [pii]
LID - 10.5935/0101-2800.20160069 [doi]
AB  - IgA Nephropathy (IgAN) is the commonest of the glomerular diseases in the world. 
      Its progression rate of 30-40% of the cases em 20-30 years makes IgAN an 
      important healthcare issue in Nephrology. Diagnosis of IgAN depends on biopsy 
      findings, particularly at immunofluorescence microscopy. The frequence of IgAN 
      diagnosis is variable in different populations and depends on screening and 
      biopsy indication policies. IgAN pathogenesis is considered multifactorial; its 
      primordial defect is the production of galactosis-deficient IgA molecules. This 
      review paper discusses the most uptodate aspects of the pathogenesis, 
      pathological classification and clinical implications of IgAN.
FAU - Soares, Maria Fernanda
AU  - Soares MF
AD  - Universidade Federal do Parana, Brazil.
LA  - por
LA  - eng
PT  - Journal Article
PT  - Review
PL  - Brazil
TA  - J Bras Nefrol
JT  - Jornal brasileiro de nefrologia
JID - 9426946
SB  - IM
MH  - Glomerulonephritis, IGA/etiology/*pathology
MH  - Humans
EDAT- 2016/12/22 06:00
MHDA- 2018/01/30 06:00
CRDT- 2016/12/22 06:00
PHST- 2016/05/09 00:00 [received]
PHST- 2016/08/08 00:00 [accepted]
PHST- 2016/12/22 06:00 [entrez]
PHST- 2016/12/22 06:00 [pubmed]
PHST- 2018/01/30 06:00 [medline]
AID - S0101-28002016000400435 [pii]
AID - 10.5935/0101-2800.20160069 [doi]
PST - ppublish
SO  - J Bras Nefrol. 2016 Dec;38(4):435-440. doi: 10.5935/0101-2800.20160069.