PMID- 30224235
OWN - NLM
STAT- MEDLINE
DCOM- 20190806
LR  - 20200701
IS  - 1873-2615 (Electronic)
IS  - 1050-1738 (Print)
IS  - 1050-1738 (Linking)
VI  - 29
IP  - 5
DP  - 2019 Jul
TI  - Ecology of the cardiovascular system: Part II - A focus on non-air related 
      pollutants.
PG  - 274-282
LID - S1050-1738(18)30190-7 [pii]
LID - 10.1016/j.tcm.2018.09.003 [doi]
AB  - An integrated exposomic view of the relation between environment and 
      cardiovascular health should consider the effects of both air and non-air related 
      environmental stressors. Cardiovascular impacts of ambient air temperature, 
      indoor and outdoor air pollution were recently reviewed. We aim, in this second 
      part, to address the cardiovascular effects of noise, food pollutants, radiation, 
      and some other emerging environmental factors. Road traffic noise exposure is 
      associated with increased risk of premature arteriosclerosis, coronary artery 
      disease, and stroke. Numerous studies report an increased prevalence of 
      hypertension in people exposed to noise, especially while sleeping. Sleep 
      disturbances generated by nocturnal noise are followed by a neuroendocrine stress 
      response. Some oxidative and inflammatory endothelial reactions are observed 
      during experimental session of noise exposure. Moreover, throughout the 
      alimentation, the cardiovascular system is exposed to persistent organic 
      pollutants (POPs) as dioxins or pesticides, and plastic associated chemicals 
      (PACs), such as bisphenol A. Epidemiological studies show positive associations 
      of exposures to POPs and PACs with diabetes, arteriosclerosis and cardiovascular 
      disease incidence. POPs and PACS share some abilities to interact with nuclear 
      receptors activating different pathways leading to oxidative stress, insulin 
      resistance and angiotensin potentiation. Regarding radiation, survivors of 
      nuclear explosion have an excess risk of cardiovascular disease. Dose-effect 
      relationships remain debated, but an increased cardiovascular risk at low dose of 
      radiation exposure may be of concern. Some emerging environmental factors like 
      electromagnetic fields, greenspace and light exposure may also require further 
      attention. Non-air related environmental stressors also play an important role in 
      the burden of cardiovascular disease. Specific methodologies should be developed 
      to assess the interactions between air and non-air related pollutants.
CI  - Copyright (c) 2018 Elsevier Inc. All rights reserved.
FAU - Argacha, J F
AU  - Argacha JF
AD  - Cardiology Department, Universitair Ziekenhuis Brussel, VUB, Belgium. Electronic 
      address: jean-francois.argacha@uzbrussel.be.
FAU - Mizukami, T
AU  - Mizukami T
AD  - Cardiology Department, Universitair Ziekenhuis Brussel, VUB, Belgium.
FAU - Bourdrel, T
AU  - Bourdrel T
AD  - Radiology Department, Imaging Medical Center Etoile-Neudorf, Strasbourg, France.
FAU - Bind, M-A
AU  - Bind MA
AD  - Department of Statistics, Faculty of Arts and Sciences, Harvard University, 
      Cambridge, MA, USA.
LA  - eng
GR  - DP5 OD021412/OD/NIH HHS/United States
PT  - Journal Article
PT  - Research Support, N.I.H., Extramural
PT  - Research Support, Non-U.S. Gov't
PT  - Review
DEP - 20180908
PL  - United States
TA  - Trends Cardiovasc Med
JT  - Trends in cardiovascular medicine
JID - 9108337
RN  - 0 (Environmental Pollutants)
CIN - Trends Cardiovasc Med. 2019 Jul;29(5):283-284. PMID: 30471986
MH  - Animals
MH  - Cardiovascular Diseases/diagnosis/*epidemiology/physiopathology
MH  - Cardiovascular System/*physiopathology
MH  - Dietary Exposure/adverse effects
MH  - Environmental Exposure/*adverse effects
MH  - Environmental Monitoring
MH  - Environmental Pollutants/*adverse effects
MH  - *Food Contamination
MH  - Humans
MH  - Noise/*adverse effects
MH  - Prognosis
MH  - Radiation Exposure/adverse effects
MH  - Risk Assessment
MH  - Risk Factors
PMC - PMC6408313
MID - NIHMS1506839
OTO - NOTNLM
OT  - Bisphenol
OT  - Coronary artery disease
OT  - Dioxins
OT  - Electromagnetic field
OT  - Endothelium
OT  - Hypertension
OT  - Noise
OT  - Oxidative stress
OT  - Pesticides
OT  - Radiation
OT  - Stroke
COIS- Disclosure of conflicts of interest: The authors declare no competing financial 
      interests.
EDAT- 2018/09/19 06:00
MHDA- 2019/08/07 06:00
PMCR- 2020/07/01
CRDT- 2018/09/19 06:00
PHST- 2018/05/24 00:00 [received]
PHST- 2018/09/03 00:00 [revised]
PHST- 2018/09/03 00:00 [accepted]
PHST- 2018/09/19 06:00 [pubmed]
PHST- 2019/08/07 06:00 [medline]
PHST- 2018/09/19 06:00 [entrez]
PHST- 2020/07/01 00:00 [pmc-release]
AID - S1050-1738(18)30190-7 [pii]
AID - 10.1016/j.tcm.2018.09.003 [doi]
PST - ppublish
SO  - Trends Cardiovasc Med. 2019 Jul;29(5):274-282. doi: 10.1016/j.tcm.2018.09.003. 
      Epub 2018 Sep 8.