PMID- 35839419
OWN - NLM
STAT- Publisher
LR  - 20240216
IS  - 2162-5220 (Electronic)
IS  - 1710-3568 (Linking)
DP  - 2022 Jun 29
TI  - When Foods Cause Itch: Clinical Characteristics, Pathophysiology, and 
      Recommendations for Food-Induced Skin and Mucosal Pruritus.
LID - 10.1097/DER.0000000000000916 [doi]
AB  - Itch occurs in various dermatologic and systemic conditions. Many patients report 
      that certain foods instigate itch, although there is limited published 
      information in dermatology on food-induced pruritus. In addition, itch severity 
      is rarely mentioned. Food can induce pruritus through either ingestion or direct 
      contact with skin or mucosal membranes. The most common type of itch provoked by 
      food is acute urticaria, often through the classical immunoglobulin E 
      (IgE)-mediated pathway. Other mechanisms include non-IgE-mediated, mixed 
      (IgE-mediated and non-IgE-mediated), T-cell-mediated, and nonimmune reactions. 
      For patients presenting with urticaria, generalized pruritus, oral pruritus, or 
      dermatitis, a thorough history is warranted, and possible food associations 
      should be considered and assessed. Although any food seems to have the potential 
      to elicit an immune response, certain foods are especially immunogenic. Treatment 
      includes avoidance of the trigger and symptom management. Careful consideration 
      should be used as to avoid unnecessarily restrictive elimination diets.
CI  - Copyright (c) 2022 American Contact Dermatitis Society. All Rights Reserved.
FAU - Vander Does, Ashley
AU  - Vander Does A
AD  - From the Dr Phillip Frost Department of Dermatology and Miami Itch Center, 
      University of Miami, FL.
FAU - Ju, Teresa
AU  - Ju T
FAU - Yosipovitch, Gil
AU  - Yosipovitch G
LA  - eng
PT  - Journal Article
DEP - 20220629
PL  - United States
TA  - Dermatitis
JT  - Dermatitis : contact, atopic, occupational, drug
JID - 101207335
SB  - IM
COIS- G.Y. conducted clinical trials or received honoraria for serving as a member of 
      the Scientific Advisory Board and consultant of Pfizer, TREVI, Regeneron, Sanofi, 
      Galderma, Novartis, Bellus, Kiniksa, and Eli Lilly, and received research funds 
      from Pfizer, Leo, Sanofi, Regeneron, Eli Lilly, and Novartis. The remaining 
      authors have no funding or conflicts of interest to declare.
EDAT- 2022/07/16 06:00
MHDA- 2022/07/16 06:00
CRDT- 2022/07/15 15:33
PHST- 2022/07/15 15:33 [entrez]
PHST- 2022/07/16 06:00 [pubmed]
PHST- 2022/07/16 06:00 [medline]
AID - 01206501-990000000-00036 [pii]
AID - 10.1097/DER.0000000000000916 [doi]
PST - aheadofprint
SO  - Dermatitis. 2022 Jun 29. doi: 10.1097/DER.0000000000000916.